Hi all.
To throw in my Saturday night, 10pm, cents...
I only skimmed the article (see above), but it seems to deal with a couple of basic problems in most decompression models.
To recap (sorry if this seems like well known facts), decompression models actually consist of two distinct models:
1) a model of the state of the body, that is how much gas dissolved where (the compartments);
2) a model of how shallow you can go before something bad happens (the decompression).
Note that the two mainline models, Buhlmann and VPM, only differ in 2). VPM uses the same compartment model as Buhlmann 16. These models are mainline because they've been published, either in a publication (Buhlmann), or as source code (VPM).
So what's wrong?
For 2), Bulhmann at least doesn't model inert mixes for decompression. There's a/b values (or M values, whichever you like) for Nitrogen and Helium separately, but nothing for mixes. And while it's okay to say that gases dissolve according to their own partial pressure only and thus independently of each other, it's not true at all when bubbles start to form. Bubbles formation still isn't so well understood. The "solution"(s) presented in M.Powell's book (which I highly recommend, BTW) are a kludge at best, with neither theoretical nor experimental support. I don't know what VPM does. That's the classic isobaric counter-diffusion (as I think it's called) problem: if you switch for an N2 (slow gas) rich to He (fast gas) rich gas, He will diffuse in faster than N2 will diffuse out, so the overall inert pP will increase. And the model doesn't tell you how high that can go. Yes, planners may warn you, but like I said, the warning algorithm isn't based on much.
For 1), they assume that compartments are directly linked to the lungs. That's a good first approximation, as blood is very fast (in that context), but it prevents modeling the ear thing that Stewart mentioned. If you have compartments in series, say fast-slow-lung, the slow compartment will continue to outgas to the fast one even tho the lungs have a lower pP because you ascended, with the same gas mix. That may lead to problems. The article seems to be saying that just compartment-blood-lungs changes things, because after a certain amount of bubble are formed, diffusion through the lungs goes down. That's not in the models either. And finally the gas composition and pressure of the blood is not constant. To wit, in arterial blood you have loads of O2 as well as N2 (especially if you're deep), but high pressure. In venous, you have much less O2 (or none), but more CO2 and N2 (on ascent), but less pressure. Again, not in the models.
Note thaht some models, DCIEM for one, have compartments in serie, not parallel like Buhlmann (DCIEM is serie only, which is going too far IMHO). But they're not published :-(
CU next week.
Matthieu